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Conclusion By integrating bioinformatics prediction with in vivo validation, this study elucidates that palmatine alleviates T2DM-associated MASLD by targeting a network of five key proteins (ADRB2, BCL3, EGR1, FOS, MAP3K8)

Pathological iron accumulation appears to result from altered expression and function of iron transport proteins at the blood-brain barrier, exacerbated by inflammatory cytokines and oxidative stress that modulate key iron transporters such as transferrin receptor, divalent metal transporter 1, and ferroportin

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However, it can be considered for individuals with both conditions

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The optimal dosing and administration of thiamine for the treatment of AD is still being investigated