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The diversity of tumor types and the complex nature of brain tissue responses to radiation underscore the limitations of a universal treatment approach

Induction of autophagy by spermidine is neuroprotective via inhibition of caspase 3-mediated Beclin 1 cleavage

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These results indicated that BME is a general tool for rapid biotinylation by TurboID in living cells

This logically suggests that therapeutic strategies designed to further enhance or sustain SYK activity following injury could potentially elevate the repair capacity above the pathological threshold, leading to improved functional outcomes