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This is thought to be due to TNF--induced hepatocyte apoptosis and can be rescued by combined loss of TNF (i.e., TBK1 / TNF / mice are viable) ( High levels IFN- or induction of IFN-stimulated genes (i.e., the IFN signature) is a remarkably consistent feature of SLE and is associated with high titers of affinity-matured autoantibodies and worse disease outcome ( IFN-Is in Protective and Pathogenic Immune Responses Among members of the IFN-I family in humans and mice, IFN- and IFN- are the best characterized and most broadly expressed