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Investigating novel therapeutic strategies for GBM is both imperative and urgently required to address the limitations of current treatment modalities [8]

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doi: 10.1158/1541-7786.MCR-18-0055 91 DixonSJPatelDNWelschMSkoutaRLeeEDHayanoMet al

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Kallistatin inhibits TGF--induced endothelial-mesenchymal transition by differential regulation of microRNA-21 and eNOS expression

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Median values are shown

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In particular, AtGRXS8 represses the transcriptional and developmental responses of the primary root to nitrate, by interfering with the activity of the TGA1 and TGA4 transcription factors (Ehrary et al

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