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In turn, Th1 cells produce IFN- and Th17 cells generate interleukin-17, which collectively contribute to sustained immune dysregulation and chronic inflammation [43,44,45,46]

The primary treatment approach for patients with MIS-C involves the use of immunomodulatory medications, including intravenous immunoglobulin (IVIG), which is a blood product containing antibodies from numerous healthy donors, as well as corticosteroids [2, 27, 28]

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Use cautiously in severe kidney impairment due to the risk of altered metabolism and accumulation of certain nutrients

Richardson, S