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Different mechanisms including DNA damage, promotion of lipid peroxidation, and protein modification are caused by oxidative stress lead to tissue damages which have been implicated in the pathogenesis of various systemic diseases

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For example, monounsaturated vaccenic acid and palmitoleic acid and a subset of polyunsaturated fatty acids were increased in both WT animals and acdh-11 mutants reared on cfa E

Earlier studies on L-proline employed the deletion method to remove L-proline from the medium used to culture cells [11] [12]

By triggering NF-B signaling, THO induces apoptotic cell death in CECs and disrupts the integrity of the corneal epithelial barrier, leading to increased permeability and susceptibility to infection [44,45]

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