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doi:10.1056/NEJMoa1301467

Anti-autophagy is one of the protective mechanisms of NKTCL because chaperon-dependent autophagy inhibitor, 17-DMAG, have been reported to suppress the cell growth of NKTCL directly [50, 51]

For instance, oxidative stress caused by the deposition of -amyloid protein (A) plays an important role in the pathogenesis of AD (Schubert et al., 1995), and the neurotoxicity of cisplatin to neuronal cells has also been confirmed and recognized (Troy et al., 2000)