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The possibility that sustained folic acid intake would deplete vitamin B-12 independent of hematopoiesis suggests a plausible alternative mechanism based on the known details of the renal handling of holoTC, whereby folic acid might interfere with holoTC reuptake or cause its sequestration in the kidney, promoting the excretion of holoTC and further depleting metabolically active vitamin B-12 during vitamin B-12 deficiency

Technically, but it wasn't designed for that purpose

A person with constipation produces hard stools that can be difficult to pass

Akpek, this includes patients with central corneal ulcers, those with significant stomal thinning with or without impending perforation or with post-infectious non-healing epithelial defects

The structural rearrangements of the host cells will likely be highly beneficial for the pathogen

Thereby, NAC can exert antioxidant and pro-osteogenic effects in vitro without specific stimulus inducing oxidative stress