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Early-phase clinical studies have demonstrated that intratumoral administration of immunoadjuvants can convert cold tumors into hot immunogenic lesions, leading to durable systemic responses in subsets of patients

24 h) promotes the migration of cultured Sprague-Dawley rat Achilles tendon fibroblasts and induces increased F-actin formation [1]

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MacFarlane, A

"Maternal vitamin B12 status and risk of neural tube defects in a population with high neural tube defect prevalence and no folic Acid fortification"

Researchers proposed that the mechanisms underlying these regenerative interactions may involve the modulation of cellular differentiation processes and the synthesis of proteins important for tissue repair in laboratory settings