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The GHK sequence also appears within the alpha-2(I) chain of type I collagen, leading to the hypothesis that collagen degradation at wound sites releases GHK fragments that then bind copper and signal for new collagen production, creating a self-repair feedback loop

Under ROS stimulation, the diselenide bond in the micelle transforms into hydrophilic selenite acid, achieving the dual effect of responsive drug release and ROS consumption

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While these pathways are theoretically relevant to soft-tissue recovery, orthopaedic-specific evidence remains minimal

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