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27 ) was utilized to compare gene expression across three distinct pair-wise tissue contrasts

Injections bypass this limitation entirely

Normobaric hyperoxia slows blood-brain barrier damage and expands the therapeutic time window for tissue-type plasminogen activator treatment in cerebral ischemia

Upon exposure to prostaglandin E 2 (PGE 2 ), an inflammatory mediator elevated in the peritoneal fluid of EMs patients, hEM15A cells further increase Cav1.3 expression

Conversely, the stained nerve sections of the CCI group displayed axonal swelling, disrupted myelin sheaths, hemorrhage between cells, and wide separation between nerve fibers

ACN effluents were diluted (1:1) with 20% ACN/0.1% FA and 50 nM of deuterated internal standards