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In GC cells, mutations in the GTP binding domain lead to sustained activation of RAS [48] and downstream PI3K/AKT/mTOR pathway, thereby promoting tumor growth and resistance to apoptosis [40] (circle marked 5 in Fig

Martin CM, Irwin N, Flatt PR, Gault VA

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they achieve the removal of lactyl groups by consuming NAD+ and generating nicotinamide (NAM) along with 2-O-lactyl-ADP-ribose (99)

This stimulation induced proliferation in hDPCs and upregulated the expression of the Wnt family members 3a, 5a, and 7a at the transcript level

Future work should focus on larger, well-designed clinical trials, deeper mechanistic validation using physiologically relevant exposure levels, improved delivery strategies, and exploration of suitable applications in food, nutraceutical, and topical formulations