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Supplementation of sodium selenite in mice protected kidneys from cadmium-induced oxidative stress and prevented apoptosis through mitochondrial pathways ( c release, and caspase activation, and prevented a decrease in voltage dependent anion channels, all of which inhibit mitochondrial signaling of cell apoptosis

Effects of zinc and selenium supplementation on thyroid function in overweight and obese hypothyroid female patients: a randomized double-blind controlled trial