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Norris EH, Giasson BI, Hodara R, Xu S, Trojanowski JQ, Ischiropoulos H, Lee VM (2005) Reversible inhibition of alpha-synuclein fibrillization by dopaminochrome-mediated conformational alterations

Chronic anemic hypoxemia attenuates glucose-stimulated insulin secretion in fetal sheep

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According to further features in preferred embodiments of the invention described below provided is a pharmaceutical composition for reducing or abolishing, extracellular or intracellular, natural or induced oxidation effects imposed on cells of an individual, the composition comprising a compound selected from the group consisting of N-acetyl cysteine ethyl ester, ,-dimethyl cysteine ethyl ester, N-acetyl-,-dimethyl cysteine, glutathione ethyl ester, N-acetyl glutathione ethyl ester, N-acetyl glutathione, N-acetyl ( ethyl ester) glutathione, N-acetyl ( ethyl ester) glutathione ethyl ester, N-acetyl glutathione amide, N-acetyl cysteine amide, N-acetyl , dimethyl cysteine amide and N-acetyl cysteine glycine amide

[1] Growth Hormone Signaling Research Studies examine how this peptide may influence growth hormone-related pathways, including pulse release behavior and timing in experimental systems