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We further demonstrate that activation of both mTOR-STAT3 and ERK-CREB pathways, induced by elevated circulation amino acids, is responsible for upregulation of Vgf and Gcg expression following glucagon receptor blockade

We then assessed the biochemical characteristics to validate the accuracy of the MZ-dock homotrimeric model generated by RoseTTAFold

Endoplasmic reticulum stress triggers an acute proteasome-dependent degradation of ATF6

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A functioning proteome allows a cell to adapt to environmental conditions and cellular stress

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