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doi: 10.1007/s00441-013-1762-z 207 SnchezB.DelgadoS.Blanco-MguezA.LourenoA.GueimondeM.MargollesA

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While some carnitine can move passively into cells, for optimal cellular function or for times of physical stress, the OCTN2 transporter is essential

We, therefore, aimed to systematically review the molecular and pathophysiological mechanisms of hyperprolactinemia and obesity, focusing on key central and peripheral pathways, supported by current experimental and clinical evidence ( Figure 1 )

APAP-induced senescence is most prevalent among cells at the perinecrotic area, where NAPQI-cys levels are high