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Here, we find EPOP can directly disrupt the dimeric form of PRC2.1 and thereby constrains its chromatin binding both in vitro and in vivo

doi: 10.1016/j.phrs.2022.106369, 135

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Disclosures: Tatjana Reic: Nothing to Disclose, Ana Visic: Nothing to Disclose, Magda Pletikosa Pavic: Nothing to Disclose, Diana Nonkovic: Nothing to Disclose, Ivona Reic: Nothing to Disclose, Anna Mrzljak: Nothing to Disclose 945 CHARACTERISTICS OF PATIENTS WITH PRIMARY BILIARY CHOLANGITIS THAT RECEIVED TRANSIENT ELASTOGRAPHY AT A TERTIARY LIVER TRANSPLANT CENTER IN NORTHERN CALIFORNIA Vishwajit Kode 1 Sylvia Sudat 2 Yinwen Wang 3 Kidist Yimam 4 , 1 California Pacific Medical Center Department of Internal Medicine, 2 California Pacific Medical Center, Center for Health Systems Research, 3 California Pacific Medical Center Research Institute, 4 California Pacific Medical Center Department of Hepatology and Liver Transplant Background: Primary Biliary Cholangitis (PBC) is a rare, chronic cholestatic disease of the small intrahepatic bile ducts of autoimmune etiology

F6P increased from an undetectable level to 2 mol/g of mycelium) (Wilson et al