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A unit of 15 g of whole-cell protein extracted was boiled with Laemmli sample buffer and resolved on a 15% polyacrylamide gel

These discrepancies likely reflect a non-linear relationship in which moderate mitochondrial dysfunction promotes migratory traits, whereas profound OXPHOS impairment constrains metastatic capacity in a tumor-type- and stage-dependent manner

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Lack of glutathione peroxidase-1 exacerbates a beta-mediated neurotoxicity in cortical neurons

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The intrinsic factor complication adds another layer