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For example, p53 stabilization results in down-regulation of numerous metabolic genes, and we observe that overexpression of RNR subunits alone is not sufficient to re-sensitize arrested cells to ferroptosis, perhaps due to the absence of upstream precursors that would allow RNR to operate at full capacity

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9,10,11,12,13,14 Of these signal transduction pathways, the PI3K-AKT-mTOR axis and Myc signaling are understood to be the primary regulators of early metabolic changes associated with T cell activation and differentiation (Fig

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