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The specific mechanism may be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis thus promote angiogenesis of gastric mucosa, and also interact with ER stress

Thus, the transition from "physiological" to "pathological" fibrosis is driven by impaired apoptosis of myofibroblasts, as well as excessive ECM deposition and accumulation, resembling a persistent wound-healing response

Cell metabolism , 28 (3), 516524.e7

Distinctive pathologic findings in proximal diabetic neuropathy (diabetic amyotrophy)

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In a study, epitalon reduced the development of mammary tumors in HEN/u2 mice and decreased the size of lung metastases [7]