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From a mechanistic perspective, the contraction of muscle fibers and the hemodynamic forces elicited by exercise activate a complex network of intracellular signaling pathways which collectively enhance mitochondrial biogenesis and quality control, stimulate angiogenic processes, and augment eNOS activity along with NO production (73, 74)

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Kunak CS, Ugan RA, Cadirci E, et al

First, pro-inflammatory cytokines can activate NOX and iNOS, leading to the production of ROS by IECs, neutrophils, and macrophages

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