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MPTP-treated mice that received schisandrin A had higher levels of TH expression in the striatum, indicating protection of dopaminergic nerve terminals (59)

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Together, this evidence suggests that while mutations or inadequacy of DNA repair mechanisms may play a role in promoting cancer neogenesis and progression, these deficiencies may increase sensitivity of cancer to therapy

Conclusion: This review outlines research directions for the development of neuroprotective plant extracts as novel PD therapies, suggesting their potential as complementary treatments alongside conventional pharmacological interventions

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wherein the presence or increase in copy number, amount, and/or activity of at least one target listed in table 1 and/or the absence or decrease in copy number, amount, and/or activity of at least one target listed in table 2, relative to a control copy number, amount, and/or activity of the at least one target, indicates that the monocyte and/or macrophage can increase its inflammatory phenotype by modulating the at least one target