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10.1038/ki.2014.372 Kidney Int

Addressing only one bottleneck leaves others unaffected

19, 1749 (2013), Abstract GSNO attenuates EAE disease by S-nitrosylation-mediated modulation of endothelial-monocyte interactions: R

This makes it harder for the liver to detoxify NAPQI, increasing vulnerability to acetaminophen toxicity even at normal doses

Is this possible

These differences in GST type and multiplicity, as well as residue changes that impact the secondary and tertiary structure of the cat and dog GST proteins, affect substrate selectivity and catalytic efficiency