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The cleavage of Corticosteroid-Binding Globulin (CBG) by neutrophil elastase is the molecular switch that releases free, biologically active cortisol precisely within the inflammatory micro-environment ( This may contribute to a state consistent with acquired local glucocorticoid resistance: the adrenal cortex may produce adequate cortisol (explaining normal serum levels), but the delivery mechanism is broken

Assertions SHALL be sufficiently unique to permit unique identification by the target RP

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Reactive oxygen species regulate a balance between mitotic catastrophe and apoptosis