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Some studies have suggested BPC-157 interacts with nitric oxide signaling pathways that regulate vascular function

Figure S11: -diversity at baseline and comparing pre- and post-intervention measurements, based on weighted UniFrac distances (left) and BrayCurtis dissimilarities (right)

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SFAs, saturated fatty acids

Furthermore, for the non- NF1 variants that have so far been observed in cNF, mechanistic studies may be valuable in determining their roles as potential drivers or functional modifiers in cNF development

Nevertheless, relative glutamatergic excess appears to be specific to early stages of illness [8], and relates to more severe symptoms at presentation [17], as well as gray matter decline [19], cognitive [20], and functional [16, 17] impairments