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This included increased protein expression of ARG-1, RAR, and TREM2, and decreased expression of iNOS [90]

Liu dng: 2 vin/ngy

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Restricting substrate influx (e.g., fatty acids or succinate) avoids over-reducing the electron carriers at the critical moment of reperfusion, and specifically blunting reverse electron transport into Complex I limits the high-amplitude ROS spikes that drive acute injury and propagate inflammatory signaling