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Obviously, VGB4 inhibited VEGF-A induced proliferation of 4T1 cells with a half maximal inhibitory concentration (IC 50 ) of ~0.37 M (Fig

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10.1038/ki.1984.143 35 GuptaS.GlezermanI

This intervention significantly suppresses the differentiation and inflammatory cytokine production (e.g., IL-17A) of pathogenic Th17 cells, while concurrently promoting the generation of Treg cells (67)