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[DOI] [PubMed] [Google Scholar] 49.Lou H, Kaplowitz N: Glutathione Depletion Down-regulates Tumor Necrosis Factor -induced NF-B Activity via IB Kinase-dependent and -independent Mechanisms

Instead, the authors showed that hyperosmotic shock leads to breakdown of sphingomyelin, accumulation of ceramide, and later on to PS exposure and cell shrinkage

The chemokine CXCL10 modulates excitatory activity and intracellular calcium signaling in cultured hippocampal neurons

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This resistance allows cancer cells to tolerate ROS accumulation and maintain survival unless the balance is further disrupted by external ROS inducers (e.g., H 2 O 2 ) or inhibitors of ferroptosis-resistance mechanisms (e.g., RSL3 and Erastin)