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One possible explanation is the constitutive nuclear activity of BACH1, a transcriptional repressor of a subset of NRF2 target genes 30 , or the lack of a positive cofactor like MAFG 31

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For now, we can only hypothesize that neuroblastomas are more sensitive to the given radiation compared to aggressive forms of glioblastoma, in which, on the contrary, this radiation promoted expansion

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