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Description

Clinical Information a familial form of congenital hyperbilirubinemia transmitted as an autosomal recessive trait

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Additionally, genetic variations in vitamin C transporters may play a role

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AbolajiAOAdedaraAOMaduJCOwaludeOTOgunyemiOMOmoboyowaDAet al

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[49] [50] Liver damage is further characterized into hepatocellular (predominantly initial Alanine transferase elevation) and cholestatic (initial alkaline phosphatase rise) types

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The animals were allowed a diet and water ad libitum

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Intriguingly, the replication track lengths were quite similar in T315I-Bcr-Abl-32D and 32D cells

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