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102 Studies have shown that mitochondrial dysfunction can lead to the occurrence of intestinal inflammation, 103,104 and excessive mitochondrial division inhibits the repair of the intestinal mucosa by disrupting butyrate metabolism in intestinal epithelial cells

ROS generated during metabolic reprogramming (e.g., enhanced glycolysis, mitochondrial dysfunction) can amplify inflammatory signals, while inflammation further exacerbates metabolic dysregulationforming a vicious cycle of metabolic abnormalitiesinflammationimmune dysregulation (111)

1 The experiment found that after adding the product and co-culturing it with skin melanocytes, it significantly inhibited melanin production by up to 26.5% compared to the control group