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To bridge this knowledge gap, future studies would need to focus on developing and validating already established clinically relevant animal models on large animals that better reflect human disease and allow for the study of long-term outcomes and tissue repair to set the stage for future human trials (Gonzalez et al., 2015

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In Alzheimers disease (AD), for example, astrocytes show abnormal glucose metabolism and reduced lactate production

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It even lowers the risk of a heart attack or heart disease if these symptoms and diseases are due to low glutathione levels