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The functional reprogramming of LCs by 1,25(OH)D is highly dependent on the integration of skin microenvironmental cues

Collectively, these findings underscore the significant potential of iron chelators in the treatment of CNS diseases, highlighting their dual role in attenuating iron-mediated toxicity and providing neuroprotection via multiple signaling pathways

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Our study has also shown that the effect of NAC on ASA treatment in GSH-depleted cells was selective in terms of recovery of GSH metabolism and oxidative stress in different cellular compartments and on mitochondrial functions and apoptosis

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